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For AD, brain tau levels as measured by PET are also being evaluated as a response biomarker, but the evidence from past clinical trials of anti-amyloid antibody treatments regarding the ability of tau-PET to reflect clinically meaningful intervention effects has been mixed (Aisen et al., 2021

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have shown that ALC supplementation (0.3, 0.6 and 1.2 mM for 5 h and 24 h) in mouse fibroblast culture media stabilizes the mitochondrial membrane, increases energy supply to the organelle and protects the developing cell from apoptosis through the mitochondrial pathway [57]

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Symptoms appear within 5 years in about 10% of patients, and within 20 years in about 20% of patients [10]