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Testosterone and other androgens upregulate IGF-1 receptor expression in skeletal muscle, which theoretically enhances IGF-1 LR3 binding efficiency studies in the Journal of Clinical Endocrinology show that androgen administration increases muscle IGF-1R density by 2040% within 24 weeks

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Glutathione does protect hepatocytes from oxidative damage during lipid metabolism, but increasing glutathione levels won't override the hormonal signals (insulin resistance, leptin resistance) that drive fat accumulation in the first place

Valproic acid is contraindicated in patients with known urea cycle disorders [seeWarnings and Precautions (5.6)]