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GPX3 can clear ROS through various signaling pathways, protecting cancer cells from damage caused by exogenous oxidants while also utilizing exogenous fatty acids provided in the TME to help OCSCs to survive in the complex TME

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This phenomenon can be explained by compounds that have little to no activity, helping the main active component reach its target through mechanisms of action that complement each other, reverse resistance, improve bioavailability, reduce metabolism and excretion, and modulate side effects (93)

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