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, the full threatening circuit in paracetamol's acute hepatic toxicity)

The clinical and public health implications of the interaction remain uncertain

biochemically, there is intracellular glutathione (GSH) depletion and decreased activity of glutathione peroxidase 4 (GPX4), lipid peroxides cannot be metabolized by the GPX4-catalyzed reduction reaction, and Fe 2+ oxidizes lipids in a Fenton-like manner, resulting in a large amount of ROS, which promotes ferroptosis 3,6

(17) found significantly decreased SOD activity levels in erythrocytes of AA patients compared with the control group