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In summary, this investigation has unveiled the existence of a newly characterised inter-organelle signalling hub in pancreatic -cells which involves active endosomal GLP-1R localisation to ERMCSs via VAPB binding and engagement of the AKAP SPHKAP to enable ERMCS-restricted cAMP/PKA activity via the assembly of PKA-RI biomolecular condensates, leading to PKA phosphorylation of the MICOS complex, enhanced mitochondrial remodelling and improved mitochondrial function, with important implications for the modulation of -cell function and survival and the potential to serve as a mechanism for GLP-1R-induced mitochondrial adaptation during metabolic stress and excess nutrient exposure

The oxidative stress is in terms of serum lipid peroxidase level and antioxidant activity in terms of superoxide dismutase and glutathione peroxidase levels were compared in type II Diabetes mellitus and age matched healthy controls

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investigation, Nusly Andres Montenegro and Sidnee Molina Gutierrez