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Because KLRG1 is widely used as a marker of highly activated, cytokine-producing ILC2s (66, 67), this finding supports the idea that melatonin drives a phenotypic transition from an activated, KLRG1 high effector state toward a more quiescent, KLRG1 low phenotype

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Staffed with a team of highly skilled technicians drawn from the University of Copenhagen, the laboratory also had close ties with Statens Serum Institut (a governmental research institute in Copenhagen)

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