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Jun 2012;147(1-3):206-16

By curbing mitochondrial ROS generation at complexes I and II, riboflavin disrupts the vicious oxidative cycle, leading to diminished lipid peroxidation, preserved mitochondrial integrity, and reduced initiation of cell death pathwaysa convergent neuroprotective mechanism across diverse neuropathological contexts (65)

From a mechanistic standpoint, mineral nutrients act not only as cofactors but also as regulators of antioxidant activity

Brain-derived neurotrophic factor and mental disorders