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Moreover, existing evidence demonstrated that inhibiting the phosphorylation of PI3K could increase AQP4 expression, which suggested that PI3K/Akt involves in the regulation of AQP4 (Song et al., 2018)

TMAO and NF-B signaling NF-B is a master regulator of inflammation, and its activation by TMAO has been extensively documented

Thus, it was reported that median IGF-I and IGFBP-5 mRNA levels in resting young muscle are more than twice higher than those in elderly muscle[354], and aging is associated with decreases in IGF-IR content and IGF-IR phosphorylation in muscle[355]