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Targeting the D1R-GHSR1a dimer for treatment of Parkinsons disease A standard treatment for Parkinsons disease typically involves the use of drugs that restore dopamine concentrations from the low levels caused by the loss of dopaminergic neurons in the substantia nigra (Mercuri and Bernardi, 2005)

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It shifts macrophages from M1 (pro-inflammatory) to M2 (reparative) phenotype and inhibits excessive NF-kB activation