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Even with decades of animal research, the human clinical data for the most popular unapproved peptides is extraordinarily thin

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[14] Local IGF-1 isoforms, mechanical loading, satellite-cell dynamics, and myostatin signaling together form the regulatory web within which any exogenous IGF-1 agonist would be acting another reason that predicting the whole-organism effect of an isolated analog from single-pathway experiments is unreliable

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