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On one hand, its a straightforward derivative of an amino acid
The metabolic-boost narrative is myth
Intriguingly, combination of BRAF V600E expression and PTEN-KD further increased EPAC2 expression but dampened EPAC1 expression (Fig
GSH depletion triggered by the Nrf2/ATF3/4/ChaC1 pathway appears to be the primary factor inducing death in drug-resistant tumor cells (138), positioning ChaC1 as a key target for certain potential anti-tumor drugs such as busatol (139) and glaucocalyxin A (140)
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