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In both intestinal and muscle tissues, we observed that kin-19 RNAi-treated animals display a similar fragmented mitochondrial network, mimicking the phenotype observed in the atp-3 control animals (Supplementary Fig

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GLP-1RAs disrupt this inflammatory cascade by diminishing adipose tissue macrophage infiltration, enhancing insulin sensitivity via reduced inflammatory signaling, and safeguarding pancreatic -cells from inflammatory harm (Pang et al