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In asthma, oxidative stress can be caused by the weakening of enzymatic and non-enzymatic antioxidant mechanisms that accompany chronic inflammations (72) ( Table 1 )

One major concern is delayed gastric emptying, a known pharmacologic effect of GLP-1 receptor agonists

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4.4 Neuroinflammation The neurotoxic environment created by synthetic cathinones activates microglia and astrocytes, leading to the release of proinflammatory cytokines, such as IL-1 and TNF-