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The activation of NOS in endothelial cells by acetylcholine, bradykinin, or adenosine is the result of the increased cytosolic Ca 2+ binding to the calmodulin subunit of the NOS complex

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Because animal growth occurs with the active production of free radicals and ATP consumption, robust antioxidative and energy transformation systems serve as the prerequisite for rapid protein gain and optimal health [25, 26]

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